[18F]FDOPA PET and clinical features in parkinsonism due to manganism
Identifieur interne : 003A55 ( Main/Exploration ); précédent : 003A54; suivant : 003A56[18F]FDOPA PET and clinical features in parkinsonism due to manganism
Auteurs : Brad A. Racette [États-Unis] ; Jo Ann Antenor [États-Unis] ; Lori Mcgee-Minnich [États-Unis] ; Stephen M. Moerlein [États-Unis] ; Tom O. Videen [États-Unis] ; Vikas Kotagal [États-Unis] ; Joel S. Perlmutter [États-Unis]Source :
- Movement disorders [ 0885-3185 ] ; 2005.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Manganèse.
English descriptors
- KwdEn :
Abstract
Manganese exposure reportedly causes a clinically and pathophysiologically distinct syndrome from idiopathic Parkinson's disease (PD). We describe the clinical features and results of positron emission tomography with 6-[18F]fluorodopa ([18F]FDOPA PET) of a patient with parkinsonism occurring in the setting of elevated blood manganese. The patient developed parkinsonism associated with elevated serum manganese from hepatic dysfunction. [18F]FDOPA PET demonstrated relatively symmetric and severely reduced [18F]FDOPA levels in the posterior putamen compared to controls. The globus pallidum interna had increased signal on Tl-weighted magnetic resonance imaging (MRI) images. We conclude that elevated manganese exposure may be associated with reduced striatal [18F]FDOPA uptake, and MRI may reveal selective abnormality within the internal segment of the pallidum. This case suggests that the clinical and pathophysiological features of manganese-associated parkinsonism may overlap with that of PD.
Affiliations:
- États-Unis
- Missouri (État)
- Saint-Louis (Missouri)
- École de médecine (Université Washington de Saint-Louis)
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Le document en format XML
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F]FDOPA PET and clinical features in parkinsonism due to manganism</title>
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F]FDOPA PET and clinical features in parkinsonism due to manganism</title>
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<series><title level="j" type="main">Movement disorders</title>
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Emission tomography</term>
<term>Manganese</term>
<term>Nervous system diseases</term>
<term>Parkinson disease</term>
<term>Parkinsonism</term>
<term>Positron</term>
<term>Positron emission tomography</term>
<term>Radionuclide study</term>
</keywords>
<keywords scheme="Pascal" xml:lang="fr"><term>Système nerveux pathologie</term>
<term>Tomoscintigraphie</term>
<term>Positon</term>
<term>Parkinsonisme</term>
<term>Exploration radioisotopique</term>
<term>Tomographie émission positon</term>
<term>Parkinson maladie</term>
<term>Manganèse</term>
</keywords>
<keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Manganèse</term>
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<front><div type="abstract" xml:lang="en">Manganese exposure reportedly causes a clinically and pathophysiologically distinct syndrome from idiopathic Parkinson's disease (PD). We describe the clinical features and results of positron emission tomography with 6-[<sup>18</sup>
F]fluorodopa ([<sup>18</sup>
F]FDOPA PET) of a patient with parkinsonism occurring in the setting of elevated blood manganese. The patient developed parkinsonism associated with elevated serum manganese from hepatic dysfunction. [<sup>18</sup>
F]FDOPA PET demonstrated relatively symmetric and severely reduced [<sup>18</sup>
F]FDOPA levels in the posterior putamen compared to controls. The globus pallidum interna had increased signal on Tl-weighted magnetic resonance imaging (MRI) images. We conclude that elevated manganese exposure may be associated with reduced striatal [<sup>18</sup>
F]FDOPA uptake, and MRI may reveal selective abnormality within the internal segment of the pallidum. This case suggests that the clinical and pathophysiological features of manganese-associated parkinsonism may overlap with that of PD.</div>
</front>
</TEI>
<affiliations><list><country><li>États-Unis</li>
</country>
<region><li>Missouri (État)</li>
</region>
<settlement><li>Saint-Louis (Missouri)</li>
</settlement>
<orgName><li>École de médecine (Université Washington de Saint-Louis)</li>
</orgName>
</list>
<tree><country name="États-Unis"><region name="Missouri (État)"><name sortKey="Racette, Brad A" sort="Racette, Brad A" uniqKey="Racette B" first="Brad A." last="Racette">Brad A. Racette</name>
</region>
<name sortKey="Antenor, Jo Ann" sort="Antenor, Jo Ann" uniqKey="Antenor J" first="Jo Ann" last="Antenor">Jo Ann Antenor</name>
<name sortKey="Kotagal, Vikas" sort="Kotagal, Vikas" uniqKey="Kotagal V" first="Vikas" last="Kotagal">Vikas Kotagal</name>
<name sortKey="Mcgee Minnich, Lori" sort="Mcgee Minnich, Lori" uniqKey="Mcgee Minnich L" first="Lori" last="Mcgee-Minnich">Lori Mcgee-Minnich</name>
<name sortKey="Mcgee Minnich, Lori" sort="Mcgee Minnich, Lori" uniqKey="Mcgee Minnich L" first="Lori" last="Mcgee-Minnich">Lori Mcgee-Minnich</name>
<name sortKey="Moerlein, Stephen M" sort="Moerlein, Stephen M" uniqKey="Moerlein S" first="Stephen M." last="Moerlein">Stephen M. Moerlein</name>
<name sortKey="Perlmutter, Joel S" sort="Perlmutter, Joel S" uniqKey="Perlmutter J" first="Joel S." last="Perlmutter">Joel S. Perlmutter</name>
<name sortKey="Perlmutter, Joel S" sort="Perlmutter, Joel S" uniqKey="Perlmutter J" first="Joel S." last="Perlmutter">Joel S. Perlmutter</name>
<name sortKey="Perlmutter, Joel S" sort="Perlmutter, Joel S" uniqKey="Perlmutter J" first="Joel S." last="Perlmutter">Joel S. Perlmutter</name>
<name sortKey="Perlmutter, Joel S" sort="Perlmutter, Joel S" uniqKey="Perlmutter J" first="Joel S." last="Perlmutter">Joel S. Perlmutter</name>
<name sortKey="Racette, Brad A" sort="Racette, Brad A" uniqKey="Racette B" first="Brad A." last="Racette">Brad A. Racette</name>
<name sortKey="Videen, Tom O" sort="Videen, Tom O" uniqKey="Videen T" first="Tom O." last="Videen">Tom O. Videen</name>
<name sortKey="Videen, Tom O" sort="Videen, Tom O" uniqKey="Videen T" first="Tom O." last="Videen">Tom O. Videen</name>
</country>
</tree>
</affiliations>
</record>
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